One of the challenges with Lyme Disease is finding a way to quantify the root causes of the various symptoms associated with it. These are often so diverse from one person to the next that there doesn’t appear to be any consistency, but there is a pattern.
Lyme disease itself was originally named after Lyme, Connecticut where a number of infections caused by the borellia bacteria started occurring. These may or may not have had something to do with a bioweapons research facility just across the water from Lyme that specialized in infecting ticks with weaponized pathogenic bacteria, but our focus here is not on fear or finger pointing but rather on understanding the pathology of this disease. Borellia is treatable by doxycycline or amoxicillin, but those antibiotics frequently fail to resolve the Lyme symptoms, and the patient and the practitioner are left to deal with a condition that they do not fundamentally understand.
This article will propose that the core symptoms of Lyme can best be understood when we examine its chemistry and parasitology. This data is drawn from my own unpublished research in advanced biochemistry, and is intended for both patients and practitioners around the world.